By R.J. Howard (Editor), N.A.R. Gow (Editor)
What makes the fungal cellphone precise between eukaryotes and what gains are shared? This quantity addresses one of the most well known and engaging aspects of questions as they pertain to the expansion and improvement of either yeast and hyphal varieties of fungi, starting with subcellular parts then mobilephone association, polarity, development, differentiation and past to the telephone biology of spores, biomechanics of invasive development, plant pathogenesis, mycorrhizal symbiosis and colonial networks. all through, structural, molecular and ecological points are built-in to shape a modern examine the biology of the fungal mobilephone.
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Extra info for Biology of the Fungal Cell (The Mycota VIII)
1990) and Ustilago maydis (Steinberg et al. 1998) has been demonstrated to require functional microtubules. For further consideration of the fungal cell cytoskeleton, the reader is referred to Chaps. 2, 5, and 6 in this volume. A comparative genomic analysis of various fungal species versus S. 1. For the most part, ER-speciﬁc functions are highly conserved. A one-to-one correspondence of genes is evident, with some exceptions. Many of these exceptions are explained by the derivation of budding yeast from a whole-genome duplication event followed by retention of duplicate gene copies with or without functional divergence (Kellis et al.
The integrity of both the ER and Golgi apparatus of animal cells requires an intact microtubule cytoskeleton. In mammalian cells depolymerization of the microtubule cytoskeleton results in the fragmentation of the Golgi apparatus (Thyberg and Moskalewski 1999) and a gradual retraction of the ER from the cell periphery (Terasaki et al. 1986). Microtubules in animal cells are generally polarized with their minus ends oriented toward the centrosome and the plus ends closer to the cell periphery. The plus end-directed microtubule motor protein kinesin is required to maintain the ER distribution, and the minus end-directed dyneins are required for maintenance of the peri-nuclear Golgi distribution (Corthésy-Theulaz et al.
2005). The A. nidulans homolog, AN11248 (GRASP55/65 homolog) shares greater homology with human GORASPs 1 and 2 than it does with Sc GRH1. Thus, some fungi may possess at least one of the genes required for the formation of Golgi stacks, though the actual function of AN11248 (and its orthologs in other fungi) remains open to investigation. These observations leave us wondering what is the functional advantage of the highly structured Golgi apparatus in higher eukaryotes? What is the penalty for the less-elaborated Golgi found in ﬁlamentous fungi?
Biology of the Fungal Cell (The Mycota VIII) by R.J. Howard (Editor), N.A.R. Gow (Editor)